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Correction to: Proteomic analysis of human synovial fluid reveals potential diagnostic biomarkers for ankylosing spondylitis
Clinical Proteomics ( IF 3.8 ) Pub Date : 2023-09-02 , DOI: 10.1186/s12014-023-09423-y
Ji-Hyun Lee 1 , Jae Hun Jung 2 , Jeesoo Kim 3, 4 , Won-Ki Baek 5 , Jinseol Rhee 6 , Tae-Hwan Kim 7 , Sang-Hyon Kim 1 , Kwang Pyo Kim 2 , Chang-Nam Son 1 , Jong-Seo Kim 3, 4
Affiliation  

Following publication of the original article [1], the authors noticed the errors in Fig. 4 and Supplementary Fig. 1.

In the Fig. 4, the CFHR5 band of G1-10 is duplicated with the C9 band.

In the Fig. 4 and Supplementary Fig. 1, the transferrin band of O6-10 is duplicated with the A6-10, independent sample O1-5.

In the Fig. 4 and Supplementary Fig. 1, the transferrin band of R6-10 is duplicated with the independent sample G1-5.

The corrected version of Fig. 4 is given below. The corrected supplementary file 5 is provided in this Correction article. The corrected figures do not alter the overall conclusions of this study, and all other data still stand. The authors regret this error and apologize for any confusion or inconvenience it may have caused.

Fig. 4
figure 4

Verification of C9, CFHR5, and MMP3 in synovial fluid by western blot. a Western blot analysis in the original synovial fluid sample set: A; AS (n = 10), R; RA (n = 10), G; gout (n = 10), and O; OA (n = 10). b Western blot analysis in the independent sample set: AS (n = 5), RA (n = 5), gout (n = 5), OA (n = 5). Transferrin was used as an input amount control

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  1. Lee JH, Jung JH, Kim JS, Baek WK, Rhee JS, Kim TH, et al. Proteomic analysis of human synovial fluid reveals potential diagnostic biomarkers for ankylosing spondylitis. Clin Proteom. 2020;17:20.

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Author notes

  1. Jihyun Lee and Jae Hun Jung equally contributed to this work.

Authors and Affiliations

  1. Division of Rheumatology, Department of Internal Medicine, School of Medicine, Keimyung University, Daegu, South Korea

    Ji-Hyun Lee, Sang-Hyon Kim & Chang-Nam Son

  2. Department of Applied Chemistry, Institute of Natural Science, Global Center for Pharmaceutical Ingredient Materials, Kyung Hee University, Yongin, South Korea

    Jae Hun Jung & Kwang Pyo Kim

  3. Center for RNA Research, Institute of Basic Science (IBS), Seoul, 08826, South Korea

    Jeesoo Kim & Jong-Seo Kim

  4. School of Biological Sciences, Seoul National University, Seoul, 08826, Korea

    Jeesoo Kim & Jong-Seo Kim

  5. Department of Microbiology, School of Medicine, Keimyung University, Daegu, South Korea

    Won-Ki Baek

  6. New drug R&D Center, ARIBIO Co. Ltd, Seongnam, South Korea

    Jinseol Rhee

  7. Department of Rheumatology, Hanyang University Hospital for Rheumatic Diseases, Seoul, South Korea

    Tae-Hwan Kim

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Corresponding authors

Correspondence to Kwang Pyo Kim, Chang-Nam Son or Jong-Seo Kim.

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The online version of the original article can be found at https://doi.org/10.1186/s12014-020-09281-y.

Below is the link to the electronic supplementary material.

12014_2023_9423_MOESM1_ESM.docx

Supplementary Material 1 S1. Verification of C4A, MBL2, and APCS in synovial fluid by western blot. (a) Western blot analysis in the original synovial fluid sample set: A; AS (n = 10), R; RA (n = 10), G; gout (n = 10), and O; OA (n = 10). (b) Western blot analysis in the Independent sample set: AS (n = 5), RA (n = 5), gout (n = 5), OA(n = 5). Transferrin was used as an input amount control.

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Lee, JH., Jung, J.H., Kim, J. et al. Correction to: Proteomic analysis of human synovial fluid reveals potential diagnostic biomarkers for ankylosing spondylitis. Clin Proteom 20, 34 (2023). https://doi.org/10.1186/s12014-023-09423-y

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中文翻译：
更正：人类滑液的蛋白质组学分析揭示了强直性脊柱炎的潜在诊断生物标志物

原文章 [1] 发表后，作者注意到图 4 和补充图 1 中的错误。

在图4中，G1-10的CFHR5带与C9带重复。

在图 4 和补充图 1 中，O6-10 的转铁蛋白条带与 A6-10、独立样品 O1-5 重复。

在图 4 和补充图 1 中，R6-10 的转铁蛋白条带与独立样品 G1-5 重复。

下面给出了图 4 的修正版本。本更正文章中提供了更正后的补充文件 5。更正后的数据不会改变本研究的总体结论，所有其他数据仍然有效。作者对这一错误表示遗憾，并对其可能造成的任何混乱或不便表示歉意。

图4
图4

通过蛋白质印迹验证滑液中的 C9、CFHR5 和 MMP3。原始滑液样本组中的蛋白质印迹分析：A ；AS（n = 10），R；RA（n = 10），G；痛风 (n = 10) 和 O; OA（n = 10）。b独立样本组中的蛋白质印迹分析：AS (n = 5)、RA (n = 5)、痛风 (n = 5)、OA (n = 5)。转铁蛋白用作输入量对照

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  1. Lee JH、Jung JH、Kim JS、Baek WK、Rhee JS、Kim TH 等。人类滑液的蛋白质组学分析揭示了强直性脊柱炎的潜在诊断生物标志物。临床蛋白质组。2020；17:20。

    文章 CAS 谷歌学术

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作者笔记

  1. Jihyun Lee 和 Jae Hun Jung 对这部作品同样做出了贡献。

作者和单位

  1. 韩国大邱启明大学医学院内科风湿科

    李智贤、金相贤、孙昌南

  2. 应用化学系，自然科学研究所，全球药物成分材料中心，庆熙大学，龙仁，韩国

    郑宰勋 & 金光杓

  3. RNA研究中心，基础科学研究所（IBS），首尔，08826，韩国

    金智秀 & 金钟瑞

  4. 首尔国立大学生物科学学院，韩国首尔，08826

    金智秀 & 金钟瑞

  5. 启明大学医学院微生物学系，大邱韩国

    白元基

  6. 新药研发中心，ARIBIO Co. Ltd，韩国城南

    李真雪

  7. 韩国首尔汉阳大学风湿病医院风湿科

    金泰焕

作者
  1. Ji-Hyun Lee查看作者出版物

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  2. Jae Hun Jung查看作者出版物

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  3. Jeesoo Kim查看作者出版物

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  4. 白元基查看作者出版物

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  8. Kwang Pyo Kim查看作者出版物

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  9. 孙昌南查看作者出版物

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  10. 金钟瑞查看作者出版物

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通讯作者

通讯作者：Kwang Pyo Kim、Chang-Nam Son 或 Jong-Seo Kim。

出版商备注

施普林格·自然对于已出版的地图和机构隶属关系中的管辖权主张保持中立。

原始文章的在线版本可以在 https://doi.org/10.1186/s12014-020-09281-y 找到。

以下是电子补充材料的链接。

12014_2023_9423_MOESM1_ESM.docx

补充材料 1 S1。通过蛋白质印迹验证滑液中的 C4A、MBL2 和 APCS。(a) 原始滑液样本组中的蛋白质印迹分析：A；AS（n = 10），R；RA（n = 10），G；痛风 (n = 10) 和 O; OA（n = 10）。(b) 独立样本集中的蛋白质印迹分析：AS (n = 5)、RA (n = 5)、痛风 (n = 5)、OA(n = 5)。使用转铁蛋白作为输入量对照。

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李，JH.，郑，JH，金，J.等人。更正：人类滑液的蛋白质组学分析揭示了强直性脊柱炎的潜在诊断生物标志物。临床蛋白质组 20 , 34 (2023)。https://doi.org/10.1186/s12014-023-09423-y

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