Abstract—Ischemia–reperfusion is a cascade of complex and interrelated pathological processes underlying many human diseases, including such socially significant diseases as stroke, myocardial infarction, acute renal failure, etc. The present review considers modern ideas about the main biochemical and signal-regulatory processes in the cell under conditions of ischemia–reperfusion. Both generally accepted and newly developed ways of ischemia–reperfusion lesion correction aimed at different chains of this pathological process are considered.

中文翻译：

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缺血再灌注损伤：发病机制的分子机制及其纠正方法

摘要：缺血再灌注是一系列复杂且相互关联的病理过程，是许多人类疾病的基础，包括中风、心肌梗死、急性肾衰竭等具有社会意义的疾病。本综述考虑了关于主要生化和信号调节的现代观点。缺血再灌注条件下细胞的过程。针对这一病理过程的不同链，考虑了普遍接受的和新开发的缺血再灌注病变校正方法。