Our viewpoint is that ethanol could act as a molecular messenger in animal and plant organisms under conditions of hypoxia or other stresses and could elicit physiological responses to such conditions. There is evidence that both animal and plant organisms have endogenous levels of ethanol, but reports on the changes induced by this alcohol at physiological levels are sparse. Studies have shown that ethanol has different effects on cell metabolism at low and high concentrations, resembling a hormetic response. Further studies have addressed the potential cellular and molecular mechanisms used by organisms to sense changes in physiological concentrations of ethanol. This article summarizes the possible mechanisms by which ethanol may be sensed, particularly at the cell membrane level. Our analysis shows that current knowledge on this subject is limited. More research is required on the effects of ethanol at very low doses, in plants and animals at both molecular and physiological levels. We believe that further research on this topic could lead to new discoveries in physiology and may even help us understand metabolic adjustments related to climate change. As temperatures rise more frequently, dissolved oxygen levels drop, leading to hypoxic conditions and consequently, an increase in cellular ethanol levels.

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动植物对生理剂量乙醇的反应：缺氧的分子信使？

我们的观点是，在缺氧或其他应激条件下，乙醇可以作为动植物有机体的分子信使，并可以引发对这种条件的生理反应。有证据表明动物和植物有机体都具有内源性乙醇水平，但有关这种酒精在生理水平上引起的变化的报道很少。研究表明，乙醇在低浓度和高浓度下对细胞代谢有不同的影响，类似于毒物兴奋反应。进一步的研究已经解决了生物体用于感知乙醇生理浓度变化的潜在细胞和分子机制。本文总结了感知乙醇的可能机制，特别是在细胞膜水平上。我们的分析表明，目前关于这一主题的知识有限。需要对极低剂量的乙醇在分子和生理水平上对植物和动物的影响进行更多研究。我们相信，对该主题的进一步研究可能会带来生理学方面的新发现，甚至可能帮助我们了解与气候变化相关的代谢调整。随着温度更频繁地升高，溶解氧水平下降，导致缺氧状况，从而导致细胞乙醇水平增加。